Short and dysfunctional telomeres sensitize the kidneys to develop fibrosis
- 19 abr 2021
- 1 min de lectura
ERA-EDTA
Telomere shortening or dysfunction may contribute to pathological, age-associated renal fibrosis by influencing the epithelial-to-mesenchymal transition (EMT) program, according to a new mouse model of kidney fibrosis.
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